Alas, due to a recent unfortunate event, I had to delay these two great interviews. But both guests were gracious enough to reschedule to this week.
How to Ask Your Question
You can ask questions of either Ivor Cummins or Joel Kahn in advance of the broadcast one of two ways:
- Comment to this blog post down below — but keep it short! (Don’t ask multiple questions)
- Comment on Twitter using the hastag: #CholesterolScience
Feel free to direct your question to the guest you want to answer. If you don’t, we’ll choose for you.
As always, keep it respectful and fun.
What causes raised homocysteine and how does that impact on disease?
I was floored when I heard Dr. Kahn and Chris Kesser “argue” about the 15% they disagree on. What about a bit of calm discussion from both on how each can interpret a study / paper differently.
Another point, I’m still looking for Dr. Kahn’s references on how sat fat affects the liver and it’s ability to clear cholesterol.
Ivor: Any idea of the mechanism behind removing calcium from arterial walls to decrease CAC?
I am one year on lchf, down 20 kg but just had CAC done and it’s 212, 60 yrs old doing all the things suggested by Ivor, how soon should I repeat the scan? My fasting bsl can still be elevated 6ish my hba1c is 31 which I think is about 4.9 in America, how can I get it down lower. I intermittent fast but don’t do extended fasts, thanks from New Zealand
Ivor: Is this too bad: TC 10.3 /LDL-C 6.7/ HDL-C 2.6/ TG 2.1
Strict keto for 3y, low carb for 5y. Always been slim, 104lb weight, very fit, middle aged woman.
CRP:0.3 – HbA1c (mmol/mol) 35
Are high TG alone a cause for concern?
I believe he’ll be taking general questions, not so much commenting on specific panels. But, you may want to check into our post on high triglycerides and that may be able to help.
Often with a bit of troubleshooting you can figure out what’s causing it, in my experience
Question for Ivor- when I consume saturated fat, my ldl p goes up into the red, but my hdl rises to a nice “healthy” level (greater than 45 but not high enough to meet lean mass hyper responder criteria.) when I lower or severely reduce my sat fat intake, yet maintain a high mono unsaturated intake, my ldl drops into the green, but hdl drops with it into the low 30s. What can be concluded from this? It’s hard to determine if that is just functional HDL rising to the occasion so to speak, and lowering when not needed as much? All other metabolic markers are excellent. Thanks for your time and insight.
Question for Joel, was the dick pick a grass fed beef sausage or an organic carrot? Thank you.
If the lipid system is primarily a means of delivering energy for LMHRs, why does substituting MUFAs for SFAs lower LDL?